Identifying new targets for the treatment of heart failure in patients with diabetic cardiomyopathy
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Initiated study of how the thrombin receptor contributes to cardiomyopathy by increasing cardiac fibrosis, especially in the setting of diabetes
Identified that inhibiting the thrombin receptor protects the heart against injury and subsequent development of cardiac fibrosis
Documented no increased activity of the thrombin receptor in the heart within the setting of experimental diabetes or high glucose conditions. Results do not suggest that diabetes worsens cardiac injury
Disseminated findings through various publications, presentations, and poster sessions
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