Informing therapeutic targets for Wisconsin patients suffering from progressive heart failure
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Found that the loss of the protein LlGl1 results in several pro-cardiac regeneration pheynotypes, including activation of the YAP transcription factor, a protein involved in the process of transcribing DNA into RNA; rapid reproduction of cardiomyocytes, cells responsible for generating contractile forces in the heart; and an increase in the number of mono-nuclear diploid cardiomycytes
Identified a node factor that may coordinate the process of cytoarchitectural modifications, the structural arrangement of neurons within the central nervous system, with cardiomyocyte proliferation
Project results have implications for promoting cardiac regeneration in non-regenerative mammals
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