Improving understanding and treatment therapies for high-risk hematological malignancies
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Gained knowledge that some subsets of T cells are more easily "programmed" to recover antitumor immunity than others
This new knowledge provides mechanistic insights for therapeutic designs that are applicable to many types of cancer
Findings from this project can be translated to clinical care rather quickly to start benefitting cancer patients locally
Awarded external funding to continue research
Published three manuscripts and presented findings internationally
Gained knowledge that the killing function of tumor reactive T cells can be restored
Began exploration of how key gene regulation machinery in tumor-reactive T cells can be exploited by genetic engineering to potentially revitalize dysfunctional T cells and enable them to kill tumor cells as a means of immunotherapy
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